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. 2017 Jul 17;8:16069. doi: 10.1038/ncomms16069

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Copyright © 2017, The Author(s)

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

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(a) H3K27me3 marks were virtually absent in the Wap-Ramp3 locus in mammary tissue from WT and ΔCDE mutants. (b) The Hoxα locus served as a control locus and showed extensive H3K27me3 coverage in mammary tissue. (c) No increase of H3K27me3 marks in ΔE and ΔCDE mutants in the cerebellum. (d) The Hoxα locus served as a control locus in the cerebellum and showed strong H3K27me3 coverage.