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. 2017 Jan 18;72(7):646–653. doi: 10.1136/thoraxjnl-2016-208789

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This is an Open Access article distributed in accordance with the terms of the Creative Commons Attribution (CC BY 4.0) license, which permits others to distribute, remix, adapt and build upon this work, for commercial use, provided the original work is properly cited. See: http://creativecommons.org/licenses/by/4.0/

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Smoking behaviour as a function of AHRR (cg05575921) methylation extent, among former and current smokers. The lower number of individuals, for smoking history compared with tobacco consumption, is explained by missing information on smoking history. Bar values are means and SEs. Estimates (95% CI) and p values per lower quintile and per 10% lower methylation extent were calculated with unadjusted linear regression. p Values for sex interaction were from a likelihood ratio test that compared the main effects in a linear regression model to a model also including a two-factor (sex by quintile of methylation extent) interaction term. AHRR, aryl hydrocarbon receptor repressor.