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. 2017 May 18;8(5):e2804. doi: 10.1038/cddis.2017.216

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Copyright © 2017 The Author(s)

Cell Death and Disease is an open-access journal published by Nature Publishing Group. This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

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ST7L mediates the effects of miR-23b on HCC progression in vivo. HCC mouse model was constructed using stable infected HCCLM3 cells using the corresponding lentiviruses. (a and b) The sizes of local liver tumors in the three groups were calculated and compared in the right diagrams. Results were represented as mean±S.D. (n=4). (c) Relative expression levels of Ki67 and ST7L were analyzed by immunohistochemistry. (d) Typical pictures for lung metastasis of HCC mouse model. (e) The percentage of mice with presence (metastatic lesion⩾1) or absence (metastatic lesion=0) of lung metastasis was calculated (n=8). *P<0.05, **P<0.01