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. 2017 Jun 1;8(6):e2842. doi: 10.1038/cddis.2017.241

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Copyright © 2017 The Author(s)

Cell Death and Disease is an open-access journal published by Nature Publishing Group. This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

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Expression analysis of AKAP1 in human cancer cells and tissues. (a–c) Immunoblot analysis of AKAP1 in human breast (a), prostate (b) and lung (c) cancer cells. Vinculin was used as a loading control. (d–f) Immunohistochemistry for AKAP1 in breast (d), prostate (e) and lung (f) high-grade (upper panels, IHC score: 2/3) and low-grade (lower panels, IHC score: 0/1) cancer tissues. Size bars are indicated in the panels. (g) IHC on glioblastoma (upper panels, IHC score 3) and astrocytoma (lower panels, IHC score 1) tissues. (h) Immunoblot analysis of AKAP1 in lysates prepared from astrocytoma and glioblastoma human tissues. Actin was used as loading control. (i) Quantitative analysis of the data shown in (h). The data are expressed as mean±S.E.M. of three experiments