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. 2017 Jun 22;8(6):e2893. doi: 10.1038/cddis.2017.266

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Copyright © 2017 The Author(s)

Cell Death and Disease is an open-access journal published by Nature Publishing Group. This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

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Physical activity prevents acute liver injury from GalN/LPS. (a) Alanine aminotransferase (ALT), (b) aspartate aminotransferase (AST) and (c) cellular-free DNA (cfDNA) measured in mice after voluntary wheel running (VWR; n=14) and in the sedentary group (SED; n=14) and 5 h after i.p. injection with GalN/LPS (each group VWR and SED n=14). Data are means±S.E.M. (d) Representative hematoxylin and eosin-stained liver sections from mice in the sedentary (SED) and voluntary wheel running (VWR) group following treatment with or without GaIN/LPS (magnification each × 400). Inflammatory cell aggregates (black arrow) and necrotic hepatocytes (white arrow) are denoted. (e) Activation of caspase 3 in the voluntary wheel running (VWR) and sedentary (SED) groups following GaIN/LPS treatment. A representative blot and densitometric analysis are provided