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. 2017 Jul 24;7:6304. doi: 10.1038/s41598-017-06683-1

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Correlations between neurophysiological outcomes and histopathology in ischaemia + vehicle, n = 6; ischaemia + short infusion, n = 6 and ischaemia + long infusion, n = 5 (neurophysiological data were not available from 1 subject in the ischaemia + long infusion group). Panels (A–C) Correlations between total seizure burden and survival of striatal calbindin-28k, calretinin and parvalbumin positive neurons. (D–F) Correlations between the day that sleep state cycling (SSC) resumed after ischaemia and survival of striatal calbindin-28k, calretinin and parvalbumin positive neurons. (G–I) Correlations between EEG power on day 7 of recovery and survival of striatal calbindin-28k, calretinin and parvalbumin positive neurons.