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. 2017 Jun 19;14(2):2081–2088. doi: 10.3892/ol.2017.6416

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Copyright: © Li et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Ad5-UPII-E1A combined with MMC or HCPT synergistically inhibits cell growth in a dose and time-dependent manner. Bladder cancer 5,637 cells treated with an increasing dose of (A) HCPT or (C) MMC and Ad5-UPII-E1A (10 multiplicity of infection) (B and D) were determined by MTT assay. Data are presented as the mean ± standard deviation of three independent experiments. *P<0.05 vs. Ad5-UPII-E1A group, #P<0.05 vs. HCPT alone group, ∆P<0.05 vs. MMC alone group. Ad5-UPII-E1A, urothelium-specific recombinant adenovirus type 5; MMC, mitomycin; HCPT, hydroxycamptothecin.