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. 2016 Sep 1;1(1):218–228. doi: 10.1089/can.2016.0013

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© Hamid Moradi et al. 2016; Published by Mary Ann Liebert, Inc.

This Open Access article is distributed under the terms of the Creative Commons License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited.

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FIG. 1. — Serum markers of renal function, BUN, and creatinine are significantly elevated in animals with IRI compared with sham-operated controls (a, b). Renal histology shows significant tubular damage at the renal corticomedullary junction in animals with IRI when compared with sham-operated controls (c, d). Damage was evaluated and expressed in the following scoring system: scoring system % damage; 0, <10%; 1, 10–25%; 2, 25–50%; 3, 50–75%; 4, 75–100%. Data are expressed as mean ± SEM, n=6–10 animals in each group ***p<0.005. BUN, blood urea nitrogen; IRI, ischemia–reperfusion injury; SEM, standard error of the mean.