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. Author manuscript; available in PMC: 2017 Jul 27.
Published in final edited form as: Eur J Pharmacol. 1993 Nov 16;249(3):265–270. doi: 10.1016/0014-2999(93)90521-i

TABLE 4.

The effect of different doses of either CPA or CPX on the average latency of neurological symptoms, degree of neurological impairment, and mortality caused by i.p. administration of NMDA.

Dose
mg/kg
Latency (s) P Impairment P T/death (h) P
30.0 NMDA 542±97 1.9±0.8 n.s. 4.7 ±0.3
1.0 CPA ±30.0 NMDA < 900 < 0.05 1.0 ± 0.0 n.s. > 5 n.s
1.0 CPX ± 30.0 NMDA 538 ±44 n.s 1.7 ± 0.1 < 0.05 > 5 n.s.
2.0 CPX ± 30.0 NMDA 406 ± 66 < 0.05 3.0 ± 0.8 < 0.05 > 5 n.s.
60.0 NMDA 414±58 6.2±0.8 2.7 ±0.8
0.5 CPA ±60.0 NMDA 467 ±51 n.s. 5.3 ± 1.1 n.s. 2.5 ± 0.6 n.s.
1.0 CPA± 60.0 NMDA > 900 < 0.05 2.1 ±0.6 < 0.05 4.7 ±0.6 < 0.05
1.0 CPX±60.0 NMDA 249±54 < 0.05 10.3±0.8 < 0.05 2.0 ±0.7 n.s
2.0 CPX±60.0 NMDA 138±28 < 0.05 11.3±0.6 < 0.05 0.8 ±0.4 < 0.05
125.0 NMDA 71 ± 5 11.5 ± 0.4 < 0.05 0.68 ± 0.5
2.0 CPA ± 125.0 NMDA 541 ± 47 < 0.05 3.1 ±0.7 < 0.05 3.65 ±0.7 < 0.05

Values are mean ± S.E.M.

Latency: delay (s) to the first observable behavioral (seizure) symptoms. T/death: time to death (h).

P: Bonferroni’s corrected Student’s t-test, n = 20/group.