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. 2017 Mar 13;6(3):e300. doi: 10.1038/oncsis.2017.3

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Copyright © 2017 The Author(s)

Oncogenesis is an open-access journal published by Nature Publishing Group. This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

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Aberrant FAM83A contributes to progression of various types of cancer. (a) Analysis of FAM83A CNV in TCGA data sets revealed the FAM83A locus is amplified in 35.5% of pancreatic cancer samples. (b) FAM83A gene CNV and corresponding mRNA expression in a TCGA pancreatic cancer data set (P<0.05, n=177). (c) FAM83A gene CNV and corresponding mRNA expression in a TCGA pancreatic cancer data set (P<0.05, n=177). (d) Kaplan–Meier analysis of overall or disease-free survival for patients with low or high FAM83A expression.