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. 2017 Jul 21;9(7):783. doi: 10.3390/nu9070783

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© 2017 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Neurofibroma resection specimen. (a,b) Spindle “wavy” cells in a matrix of fine fibrillary collagen; neurofibromatous tissue merges with mature fat and ectatic vessels (Hematoxylin-eosin, Magnification 10×); (c,d) Spindle cell nuclei in a fine fibrillary matrix, at a greater magnification (Hematoxylin-Eosin, Magnification 40×); (e) S100 immunohistochemical test confirmed the neurogenic origin of the lesion (Magnification 20×); (f) Actin-hhf35 immunohistochemical test excluded the myogenic origin of the lesion (note that only perivascular splindle smooth cells were reactive) (Magnification 20×); (g) Proliferation index by Ki67 immunohistochemical test was almost negative (Magnification 20×).

Neurofibroma resection specimen. (a,b) Spindle “wavy” cells in a matrix of fine fibrillary collagen; neurofibromatous tissue merges with mature fat and ectatic vessels (Hematoxylin-eosin, Magnification 10×); (c,d) Spindle cell nuclei in a fine fibrillary matrix, at a greater magnification (Hematoxylin-Eosin, Magnification 40×); (e) S100 immunohistochemical test confirmed the neurogenic origin of the lesion (Magnification 20×); (f) Actin-hhf35 immunohistochemical test excluded the myogenic origin of the lesion (note that only perivascular splindle smooth cells were reactive) (Magnification 20×); (g) Proliferation index by Ki67 immunohistochemical test was almost negative (Magnification 20×).