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. 2017 May 3;118(2):949–963. doi: 10.1152/jn.00772.2016

Fig. 4.

Fig. 4.

When activated by POC stimulation, MCN1 intraburst firing rate is not regulated by circuit feedback. Ai: when triggered by POC stimulation, MCN1 activity is regulated by pyloric-timed AB inhibition during both the protraction and retraction phases. MCN1 was recorded extracellularly in the ion. Aii: in the absence of circuit feedback, pyloric-timed interruptions in MCN1 firing were eliminated. B: across preparations, there was no difference between the MCN1 intraburst firing rate during the protraction and retraction phases of POC-triggered gastric mill rhythms (n = 8) or its average firing rate in the absence of circuit feedback (n = 6). Pyloric-timed interruptions in firing during protraction and retraction were not included in the intraburst firing rates. nsP > 0.05.