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. 2017 Aug 1;8:42. doi: 10.1186/s13229-017-0160-x

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — Pathways altered from prenatal VPA in both transcriptomic and proteomic samples. Individual pathway heat maps of differential gene expression between saline and VPA amygdala samples at P10 (a, d), P21 (b, e, g, i, k, m), and of differential protein expression at P21 (c, f, h, j, l, n) depict log2 fold changes of genes with p < 0.05. Changes in activation (z-scores) between groups are noted for pathways in which changes were predicted. PKA protein kinase A, GDI guanine nucleotide dissociation inhibitor