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Carrageenan-induced AMPAr trafficking is NMDA receptor dependent. (A) Pretreatment with MK801, an NMDA receptor antagonist, blocks inflammation –induced trafficking of GluA1 to the plasma membrane. (B) MK801 treatment had no affect on GluA2 at this time. Data is presented as mean±SEM, * n=4, p < 0.05, and *** p < 0.001
