Figure 3.

Collateral enlargement in topographical relation (satellite) with severe stenosis or occlusion. A double occlusion of LAD (anterior view) and occlusion of RCA (posterior view) apparently compensated by enlarged collaterals in a non cardiac patient dead from brain hemorrhage. B, similar condition in cases with RCA occlusion (arrow) without corresponding myocardial infarct with numerous homo and intercoronary collaterals of the anterior wall (C), and (D) septum. Occlusion of LAD without evidence of other stenotic changes of the coronary arteries in a 39-year-old woman with rheumatic heart disease and mitral insufficiency. In this case, arteritis was documented histologically by sampling before corrosion. An acute infarct (avascular area at the apex, arrow) was present. F, a single, high enlarged collateral from LCX, supplying the distal tract of an occluded LAD. Note, numerous normal anastomoses. This indicates that ischemia is not the cause (no diffuse enlargement of all collaterals in the whole ischemic area) but rather pressure gradient induces selective compensatory routes.