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. 2017 Aug 3;8:182. doi: 10.1038/s41467-017-00274-4

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 9 — Mitochondrial elongation is required for longevity in diverse mutants. a–i Survival curves for adults of the indicated genotypes, overexpressions (oe), and RNAi treatments. The wild-type and eat-3(RNAi) survival curves are shown in graphs for comparison. eat-3 RNAi significantly reduced the extended lifespans relative of all tested genotypes, overexpression, and RNAi treatments except for vhl-1 mutants and wild-type control e. All lifespan experiments were performed with four biological replicates. See Supplementary Table 2 for statistics