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. 2017 Jul 10;6(7):e356. doi: 10.1038/oncsis.2017.59

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Copyright © 2017 The Author(s)

Oncogenesis is an open-access journal published by Nature Publishing Group. This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

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Inverse correlation between ASCT2 and miR-137 expression in clinic tumor specimens. (a) Relative levels of miR-137 (left panel) and ASCT2 (right panel) in normal colonic epithelial NCM460 cells and colorectal carcinoma cell lines analyzed by real-time qPCR and immunoblot assays. Actin was used as a loading control. (b, c) Relative levels of miR-137 (b), analyzed by real-time qPCR, and ASCT2 (c), analyzed by immunoblot, in twelve pairs of normal colorectal tissues and colorectal carcinoma specimens. (d–g) Correlation between miR-137 and ASCT2 in colorectal carcinoma (d), glioblastoma (e), pancreatic (f) and prostate cancer (g), performed by Pearson’s correlation analysis.