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. 2017 Aug 3;7:7222. doi: 10.1038/s41598-017-07833-1

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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GA blocked the exCa²⁺ influx to stabilize RBL-2H3 through reducing calcium channel proteins expression. ^* P < 0.05, significantly different from control and ^# P < 0.05, significantly different from sensitization control without GA treatment (n = 3). (A) Effect of GA on [Ca²⁺]_i. (B) The mRNA relative expression of Orai1, STIM1, TRPC1 and IP3R. (C) The protein expression of calcium channel proteins.