| NUCL |
Abundant in exponentially growing cells Cytoplasmic and surface NUCL in gliomas Surface NUCL is dependent on association with actin cytoskeleton Cell-surface NUCL is a novel angiogenesis marker |
[33, 68–70] |
| VIM |
Role in formation of lamellipodia and invadipodia during cell invasion and migration Typical for infiltrative gliomas with poor prognosis Marker for epithelial to mesenchymal transition Differentially expressed in glioblastomas |
[39, 71–73] |
| NAP1L1 |
Human NAP1L1 locates within the nucleus of dividing cells Co-existence of NAP1L1 and NAP1L4 in neural stem cells and neurons Transcriptional regulation NAP1L1 is overexpressed in small-intestinal carcinoids |
[74–78] |
| TUFM |
Chaperone-like properties in protein folding and renaturation under stress conditions Translational expression of mitochondrial DNA Involved in head and neck squamous cell carcinoma Overexpressed in colorectal carcinoma |
[79–81] |
| DPYSL2 |
Highly expressed during brain development, rarely in post-developmental brain Over-expressed in colorectal carcinoma Altered expression in glioblastomas Promoter of microtubule assembly and neuronal development |
[38, 71, 82, 83]] |
| CRMP1 |
Decreased in glioblastomas expressing EGFRvIII Invasion suppressor gene in lung cancer Associated with microtubule-containing structures in mitotic cells |
[38, 84, 85] |
| ALYREF |
Transport of fully processed mature mRNA out of the nucleus NUP107 complex (consisting of ALYREF, SNRPE, NUP107 and NUP50) is altered in 19% of glioblastomas |
[37, 86] |
