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. 2017 Jul 6;16(3):2538–2546. doi: 10.3892/mmr.2017.6940

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Copyright: © Zhang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 7. — MEPM cellular proliferation rate was decreased in atRA-treated embryos. (A) Ki-67 expression in palatal shelves isolated from control and atRA-treated embryos between E12.5 and E14.5. Scale bars, 20 µm. (B) Protein expression levels of p-ERK, total ERK1/2, p21, cyclin D1 and Ki-67 in MEPM cells isolated from control and atRA-treated embryos (n=3/group). *P<0.05 vs. Con group. MEPM, mouse embryonic palate mesenchymal; atRA, all-trans retinoic acid; E, embryonic day; p, phosphorylated; ERK, extracellular signal-regulated kinase; Con, control.