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. 2017 Jul 18;38(3):1473–1481. doi: 10.3892/or.2017.5831

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Copyright: © Ji et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — Histology of the control and MNNG-induced rat gastric tissues. (A) Superficial gastritis was noted in the MNNG-12 group, atrophic gastritis in the MNNG-24 group and the cardiac part of the MNNG-36 group, and atypical hyperplasia in the pyloric part of the MNNG-36 and MNNG-48 groups. (B) H&E staining of ulcers in the MNNG-36 group. (C) H&E staining of gastric tissues in the MNNG-36 group. Intestinal metaphase changes were observed. Goblet cells were noted in the gastric mucosa. (D) H&E staining for gastric tissuse in the MNNG-48 group. Esophageal-like sarcoma lesions were observed. (E) Small intestinal type metaplasia was detected using AB-PAS staining. MNNG-12, 24, 36 and 48 represent the rats treated with MNNG for 12, 24, 36 and 48 weeks, respectively. Original magnification of the left panel, ×100; the right panel, ×200; scale bar, 50 µm.