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. 2016 Apr 4;14(8):675–684. doi: 10.1038/cmi.2015.111

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Copyright © 2016 Chinese Society of Immunology and The University of Science and Technology

This work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by-nc-nd/4.0/

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Depletion of NKT cells attenuates liver injury induced by CpG in HBs-Tg mice. HBs-Tg mice were treated with control IgG, anti-NK1.1 mAb (PK136) or anti-ASGM1 24 h before CpG injection. Blood and liver tissue were collected 24 h after CpG injection. Liver injury was evaluated by (a) serum ALT and (b) H&E staining of liver tissue (original magnification, × 200). Data are representative of three independent experiments and are shown as the mean±s.e.m. (n=6 per group). **P<0.001. ALT, alanine aminotransferase; HBs-Tg, hepatitis B surface antigen transgenic; H&E, hematoxylin and eosin; IgG, immunoglobulin G; mAb, monoclonal antibody; NKT, natural killer T; NS, not significantly different.