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. 2017 Jul 17;13:1744806917723789. doi: 10.1177/1744806917723789

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© The Author(s) 2017

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (http://www.creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 3. — N-Methyl-D-aspartate (NMDA)-evoked current responses in remifentanil and inhibitors treated dorsal horn neurons. Significant increases in NMDA peak current amplitude occurred with remifentanil exposure. There was a significant decrease in NMDA-evoked current after chelerythrine, KN93, and PD98059 treatment.*p < 0.05 versus time 0 in group Remi, ^#p < 0.05 group KN93 versus group Remi, ^&p < 0.05 group chelerythrine versus group Remi, ^ap < 0.05 group PD98059 versus group Remi.