Skip to main content
. 2001 Jul 31;98(17):9671–9676. doi: 10.1073/pnas.161151798

Figure 2.

Figure 2

p53, p33ING1b, and p33ING2 protein expression, and p53 acetylation after DNA damage (A–E). After cells were treated with 10 Gy of γ-irradiation (IR), 0.2 μg/ml doxorubicin (DOX), 30 μg/ml etoposide (ETO), 0.3 μg/ml neocarzinostatin (NCS), 0.03 units/ml bleomycin (BLEO), or 2 μg/ml cis-platinum (CDDP), protein expression of p53, p33ING1b, p33ING2, and actin and p53 acetylation at Lys-382 at each time point were analyzed by Western blotting. (F) The steady-state level of each protein was quantified by densitometry and normalized with actin. p53, p33ING1b, p33ING2, and p53 acetylation at Lys-382 after exposure to ETO.

HHS Vulnerability Disclosure