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. 2017 Aug 10;7:7744. doi: 10.1038/s41598-017-08082-y

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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SNAP-25b-deficient islets secrete more insulin than wild-type littermates in response to glucose. Glucose-induced insulin release was measured in isolated pancreatic islets from male and female 12 week old wild-type (WT) and SNAP-25b-deficient (MT) mice (a). AUC was calculated for the first (b) and second (c) phase of insulin secretion as well as for KCl stimulation (d). n = 7–8 animals per group. WT mice, black circles and bars; MT mice red circles and bars. Data are represented as mean ± SEM, *P < 0.05, **P < 0.01, ***P < 0.001.