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. 2017 Aug 11;7:7980. doi: 10.1038/s41598-017-08792-3

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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A general survey of NTP treatment affection on triple-negative breast carcinoma and normal cells through different cellular pathways.(A) NTP treatment inhibits proliferation of MDA-MB-453 and MCF10A cells through ROS-dependent cellular pathway (not discussed in this article). (B) NTP treatment effects on reduction of proliferation through IL-6R/pSTAT3 pathway in MDA-MB-231 and MCF10A cells. (C) NTP treatment effects on recovering the proliferation especially in normal breast MCF10A cells with post-treated 5 mM NAC.