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. 2017 Aug 11;7:7894. doi: 10.1038/s41598-017-08480-2

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Improved whole-body glucose homeostasis in exercise-trained Atg7^h&mKO mice. (a) Overnight fasting blood levels of glucose, insulin, and ketones, and resulting HOMA-IR (n = 5–11). (b) GTT and respective area under the curve (AUC) (n = 8–11). (c) ITT (n = 8–11). (d) Liver mass normalized to tibia length (TL), and liver triglyceride (TG) content (n = 5–11). (e) mRNA of hepatic genes involved in lipid homeostasis and inflammation (n = 5–9). *P < 0.05, **P < 0.01 and ***P < 0.001 in comparison to all other groups; ^# P < 0.05, ^## P < 0.01 in comparison to Atg7^fl/fl-HFD and Atg7^fl/fl-HFD&EX groups; and ^Φ P < 0.05, ^ΦΦ P < 0.01 in comparison to respective HFD only, sedentary group.