Table S1.
Correct HCM with hypertension (n=30) | Description |
---|---|
Female, born 1922, died 2015 | Septal and midseptal hypertrophy 17 mm, ECG repolarization abnormalities, maximal LVOT gradient 68 mmHg, mild controlled hypertension |
Female, born 1924, died 2013 | Septal hypertrophy 18 mm, mean gradient 58 mmHg, pacemaker due to reduced obstruction, ECG repolarization abnormalities, mild controlled hypertension |
Female, born 1926, died 2015 | Septal hypertrophy 17 mm, Vmax 2.8 m/s, ECG repolarization abnormalities, mild controlled hypertension with a short history |
Female, born 1928, died 2016 | Septal and midseptal hypertrophy 20 mm, Vmax 3.8 m/s, mild controlled hypertension |
Male, born 1931, died 2013 | Septal hypertrophy 17 mm, MYBPC positive, mild hypertension |
Female, born 1931, died 2016 | Septal hypertrophy 20 mm, Vmax 4.2 m/s, mild controlled hypertension, heredity for sudden death |
Female, born 1933, died 2013 | Septal hypertrophy 17 mm, Vmax 2.6 m/s, unexplained syncope, ECG repolarization abnormalities, mild controlled hypertension |
Female, born 1937, died 2012 | Septal, midseptal hypertrophy 21 mm, ECG repolarization abnormalities, mild controlled hypertension |
Female, 95 years | Septal hypertrophy 22 mm, ECG repolarization abnormalities, mild controlled hypertension |
Male, 89 years | Apical hypertrophy, not reported maximal thickness, ECG repolarization abnormalities, mild controlled hypertension |
Female, 85 years | Septal-apical hypertrophy 17 mm, grandmother sudden death at 56 years, MYBPC, mild controlled hypertension |
Female, 83 years | Inferolateral hypertrophy 19 mm, ECG repolarization abnormalities, mild controlled hypertension |
Female, 81 years | Septal hypertrophy 20 mm, 2.3 m/s, ECG repolarization abnormalities, mild controlled hypertension |
Female, 78 years | Septal hypertrophy 26 mm, 4.1 m/s, myectomy, ECG repolarization abnormalities, mild controlled hypertension |
Female, 76 years | Septal hypertrophy 19 mm, 3.7 m/s, ECG repolarization abnormalities, mild controlled hypertension |
Male, 74 years | Septal-midseptal hypertrophy 24 mm, 2.4 m/s, ECG repolarization abnormalities, mild controlled hypertension |
Male, 74 years | Septal hypertrophy 26 mm, myectomy, genopositive, ECG repolarization abnormalities, mild controlled hypertension |
Female, 74 years | Septal hypertrophy 18 mm, 3.1 m/s, ECG repolarization abnormalities, mild controlled hypertension |
Female, 74 years | Septal hypertrophy 21 mm, 2.2 m/s, ECG repolarization abnormalities, mild controlled hypertension |
Female, 73 years | Septal hypertrophy 19 mm, 4.4 m/s, ECG repolarization abnormalities, mild controlled hypertension |
Female, 73 years | Septal hypertrophy 22 mm, 4.8 m/s, ECG repolarization abnormalities, mild controlled hypertension |
Male, 71 years | Septal hypertrophy 21 mm, 2.9 m/s, ECG repolarization abnormalities, mild controlled hypertension |
Male, 71 years | Septal hypertrophy 23 mm, myectomy, 5.6 m/s, ECG repolarization abnormalities, mild controlled hypertension |
Female, 71 years | Septal hypertrophy 24 mm, myectomy, sudden death of two cousins, ECG repolarization abnormalities, mild controlled hypertension |
Female, 70 years | Posterior hypertrophy 19 mm, ECG repolarization abnormalities, mild controlled hypertension |
Female, 66 years | Septal hypertrophy, myectomy, MYH7 positive, ECG repolarization abnormalities, mild controlled hypertension |
Male, 60 years | Septal hypertrophy 23 mm, ECG repolarization abnormalities, mild controlled hypertension |
Male, 57 years | Septal hypertrophy 22 mm, ECG repolarization abnormalities, mild controlled hypertension |
Male, 57 years | Septal hypertrophy 20 mm, mean gradient 80 mmHg, ECG repolarization abnormalities, mild controlled hypertension |
Male, 54 years | Septal hypertrophy 19 mm, maximum gradient 39 mmHg, unexplained syncope ECG repolarization abnormalities, mild controlled hypertension |
Abbreviations: ECG, electrocardiogram; HCM, hypertrophic cardiomyopathy; LVOT, left ventricular outflow tract; MYBPC, myosin binding protein C; Vmax, velocity maximal.