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. 2017 Aug 15;8:244. doi: 10.1038/s41467-017-00279-z

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — Genome-wide analysis of potential PtpA-binding sites in U937 cells. a Genomic distribution of potential PtpA-binding regions in U937 cells. b Biotype distribution of potential PtpA-binding regions in U937 cells. c Distribution of the chromosomal location of PtpA-specific ChIP-seq signals (fold-enrichment > 10). d The number and position of the potential PtpA-binding sites along 20 kb from the nearest TSS of the ncRNAs or protein-coding genes, respectively. e PtpA consensus motifs defined by MEME motif analysis of the sequences of PtpA ChIP-seq peaks. P values were calculated by TOMTOM match statistics. “% of targets” represents the percentage of motif-containing fractions in potential PtpA targets. The size of each letter was proportional to the frequency of each nucleotide in that position within the consensus motif