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. 2017 Aug 15;7:8267. doi: 10.1038/s41598-017-07434-y

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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CXCL10 transcripts and protein are induced after pegIFNα treatment in HEV infected mice. RNA was isolated from non-treated and 2 weeks pegIFNα treated HEV gt1, gt3 and HBV infected mouse livers and was analyzed for the expression of human CXCL10 (a). X-axes indicate treatment dosage and virus genotype (a). Given values on y-axes are RNA levels in fold changes over uninfected non-treated mice (a). Human CXCL10 levels were measured using ELISA in mouse serum of uninfected, HEV-infected and HEV-infected pegIFNα treated mice (b). Dotted line indicates lower limit of detection (LLOD) (b). Gray bars indicate HEV gt1, black bars HEV gt3 and striped bar uninfected (a + b).