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. 2017 Aug 15;8:255. doi: 10.1038/s41467-017-00151-0

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — The state of histone H4 acetylation changes in cells exposed to acute stress. a Genome browser images of heat-induced (DNAJB1) and heat-repressed (SNHG8) genes, showing the histone H4 acetylation (black) and transcription from plus (red) and minus (blue) strands prior to (NHS) and upon (HS) heat stress. b Average ChIP-seq intensity of histone H4 acetylation at promoters (upper panels) and gene bodies (lower panels) of genes grouped by their transcriptional response. c The change in histone H4 acetylation upon stress at individual promoters of up- and downregulated genes. The grey dashed line in (b, c) marks the +500 nt position from TSS. d Genome browser images of histone H4 acetylation at dTREs with upregulated (left) and downregulated (right) Pol II density upon stress. e The change in histone H4 acetylation upon stress at individual up- or downregulated dTREs