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. 2017 Aug 15;7:8188. doi: 10.1038/s41598-017-07607-9

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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(A) Exemplar data Subject 3, each color represents a single community left hemisphere lateral view. There was marked fragmentation of the inferior frontal and middle-temporal networks with relative disconnection between the frontal and subcortical regions, with the left hemisphere grouping into 9 modules. Subject 3 had a lesion volume of 99.24 cm³, percent white matter damage of 0.096, and a WAB-AQ score of 58.2. There was unstable clustering and missing nodes in the community affiliation matrix compared to the right hemisphere matrix. (B) Exemplar data Subject 3, right hemisphere lateral view, each color represents a single community. There was no apparent fragmentation, and the network was divided into 4 communities. The community affiliation matrix showed relatively stable clustering over 100 runs.