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. 2017 Jun 9;16(2):1117–1124. doi: 10.3892/mmr.2017.6744

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Copyright: © Du et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — Immunohistochemical staining for α-SMA in lung tissue sections isolated from newborn Control and PPHN model rats. Representative lung histological sections of (A and B) control and (C and D) PPHN are shown. An increase in medial thickness in pulmonary arteries in (C) PPHN group compared to (A) control. When compared with the (B) control group, the distal pulmonary arterioles were greatly increased in the (D) PPHN group. Magnification, ×40; scale bar, 50 µm; red arrows indicate pulmonary arterioles (in A and C) and distal pulmonary arterioles (in B and D). α-SMA, α smooth muscle actin; PPHN, persistent pulmonary hypertension of the newborn.