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. 2017 Aug 18;7:8802. doi: 10.1038/s41598-017-09438-0

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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The circadian rhythms of salivary IgA secretion in SCN-lesioned mice. (a,b) Representative double-plotted actograms of locomotor activity for one sham mouse and one SCNX mouse after pilocarpine (a) or NE administration (b). (c,d) Representative Chi-squared periodogram analysis of behavioral data for one sham mouse and one SCNX mouse after pilocarpine (c) or NE (d) administration. Straight lines in each figure indicate statistical significance. (e,f) Salivary IgA concentration rhythms in non-SCN-lesioned (sham) and SCN-lesioned (SCNX) after pilocarpine (e) or NE administration (f) (n = 4–6). Values are shown as mean ± SEM. (f) ^$$p < 0.01, sham group (one-way ANOVA). **p < 0.01, *p < 0.05 vs. sham ZT18. ^#p < 0.05 vs. sham ZT0.