Skip to main content
. 2017 Aug 7;2017:5841814. doi: 10.1155/2017/5841814

Figure 6.

Figure 6

EA treatment suppressed the level of inflammatory cytokines in the downstream of TLR4 signaling pathway posttrauma. TNF-α, IL-1β, and IL-6 expression in the hippocampus were determined by ELISA. (a–c) Quantitative analysis showed that three inflammatory cytokines were elevated in the TBI group compared with the sham and sham + EA groups. EA treatment induced significant abatement of these cytokines in the TBI + EA treatment. Reversely, LPS administration abrogated the inhibitory effect of EA on TLR4-mediated inflammation in the TBI + EA + LPS group. There was no significant difference in these inflammatory cytokine expression between the TBI + EA + Veh and TBI + EA groups. ∗P < 0.05 versus the TBI group, †P < 0.05 versus the TBI + EA + LPS group.