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. 2017 Aug 22;7:371. doi: 10.3389/fcimb.2017.00371

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Copyright © 2017 Martin, Leyton, Hott, Arancibia, Spichiger, McNiven, Court, Concha, Burgos and Otth.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Neuronal response to HSV-1 infection lacking the HSV-1 tegument protein VP11/12. (A) Cortical primary neurons were infected with either HSV-1 wild type or a deleted HSV-1 version that lacks the viral gene UL46 (HSV-1 ΔUL46). After 4, 8, and 18 h post-infection (hpi) equivalent amounts of cell extracts were subjected to SDS-PAGE followed by Western blotting and normalized levels of p-Src/Src (B), p-Dyn2/Dyn2 (C), and ICP8/β-tubulin (D) determined. Bars represent the mean ± SD of three independent experiments. ^****p < 0.0001, ^***p < 0.001, ^**p < 0.01, ^*p < 0.05, Brackets display treatment comparisons.