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. 2017 Aug 29;7:9686. doi: 10.1038/s41598-017-09671-7

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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IL-6, TNFα and RANKL expression were increased in the hypertrophic gastric mucosa of PDP patients. P1, patient1; P4, patient4; P6, patient6; C, normal control. a–d, e–h and i–l showed immunohistochemistry staining of IL-6, TNFα and RANKL respectively. IL-6, TNFα and RANKL expression were stronger in hypertrophic gastric mucosa of PDP patients (P1 a,e,i, P4 b,f,j, P6 c,g,k) than that of healthy control (C d,h,l). IL-6, TNFα and RANKL expression were strongest in patient1 and gradually decreased in patient4 and patient6. Immunostained specimens were assessed by pathologists blinding to clinical features.