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. 2017 Jun 14;142(3):444–455. doi: 10.1111/jnc.14071

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Published 2017. This article is a U.S. Government work and is in the public domain in the USA.

This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Corticosterone (CORT) pretreatment exacerbates chlorpyrifos oxon (CPO)‐induced neuroinflammation. Effects of CPO exposure (8 mg/kg, i.p.) with and without prior CORT treatment (400 mg/L, 1.2% EtOH) on neuroinflammation as measured by qPCR of inflammatory cytokines and chemokines at 6 h post‐CPO. Tumor necrosis factor‐alpha (TNFα), IL‐6, (C–C) chemokine ligand 2 (CCL2), IL‐1b, leukemia inhibitory factor (LIF), and oncostatin M (OSM) were measured in cortex (left panels) and hippocampus (right panels). Data represents mean ± SEM (N = 4–6 mice/group). Statistical significance of at least p ≤ 0.05 is denoted by * compared with relevant control (vehicle or CORT) and # compared with treatment (saline or CPO).