Effects of imidacloprid on AMPKα pathway and TNFα
expression
in white adipose tissue. Mice were fed with low or high fat diet supplemented
with imidacloprid (0, 0.06, 0.6, and 6 mg/kg bw/day) for 12 weeks.
(A) CaMKKβ, Ca2+/calmodulin-dependent protein kinase
β; (B) pAMPKα/AMPKα, phosphorylated AMP-activated
protein kinase-α/AMP-activated protein kinase-α; (C) pACC/ACC,
phosphorylated acetyl-CoA carboxylase/acetyl-CoA carboxylase; (D)
representative pictures; (E) TNFα, tumor necrosis factor α.
Numbers represent mean ± SE (n = 4–6).
Means with different letters are significantly different at P < 0.05.