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Influence of AMPKα activation on adipogenesis induced by imidacloprid in 3T3-L1 adipocytes. (A) Triglyceride; (B) representative pictures; (C) C/EBPα, CAATT element binding protein-α; (D) pAMPKα/AMPKα, phosphorylated AMP-activated protein kinase-α/AMP-activated protein kinase-α. Cells were treated with imidacloprid (10 μM) or AICAR (40 μM) for 8 days. Numbers represent mean ± SE (n = 3). Means with different letters are significantly different at P < 0.05.
