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. Author manuscript; available in PMC: 2017 Aug 31.
Published in final edited form as: Brain Res. 2016 Apr 27;1643:140–151. doi: 10.1016/j.brainres.2016.04.063

Fig. 4.

Fig. 4

SAL mitigated markers of neuroinflammation after single blast. We measured a significant increase in NFκB p65 expression at 24 h post-sTBI (*P<0.05 vs Vehicle); SAL significantly mitigated NFκB p65 expression (#P<0.05 vs sTBI+Vehicle) (A). We revealed a significant increase in iNOS protein expression at 24 h post-sTBI (*P <0.05 vs vehicle); SAL significantly mitigated iNOS expression (#P<0.05 vs sTBI+Vehicle) (B). We measured a significant increase in IL-1β mRNA abundance at 24 h post-sTBI (*P<0.05 vs Vehicle); SAL significantly mitigated IL-1β abundance (#P<0.05 vs sTBI +Vehicle) (C). We measured a significant increase in TNFα mRNA abundance at 24 h post-sTBI (*P<0.05 vs Vehicle); SAL significantly mitigated TNFα abundance (#P<0.05) (D). One-way ANOVA, Newman-Keul’s post hoc. Mean±S.E.M. n=4.