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. 2017 Aug 31;7:10269. doi: 10.1038/s41598-017-10108-4

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Functional difference between arterial and venous VSMCs in vitro. Reduction in proliferation rate was limited to VSMCs isolated from RP105^−/− veins. Decrease in migration of VSMCs isolated from RP105^−/− mice was restricted to arterial cells only. Proliferation rate and migration were measured over 16 h time period. (b) Venous VSMCs isolated from WT and RP105^−/− mice produce significantly higher amounts of inflammatory cytokines IL6 and MCP1. Cells were maintain in culture for 14 days. ^*P < 0.05; n = 3.