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. 2017 Aug 31;7:10269. doi: 10.1038/s41598-017-10108-4

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Effect of RP105 deficiency on macrophage function. (a) Bone marrow-derived macrophages from RP105 primed towards pro-inflammatory phenotype secrete increased levels of MCP1 and IL6 as compared to WT control mice. (b) Anti-inflammatory macrophages from RP105 secrete increased levels of repair associated cytokine IL10. ^*P < 0.05; n = 3.