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. 2017 May 2;369(3):445–454. doi: 10.1007/s00441-017-2626-8

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© The Author(s) 2017

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license and indicate if changes were made.

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Fig. 4 — MPO expression in hippocampus from AD patients and control cases. Examples of MPO-immunoreactive cells in the CA1 region (a), dentate gyrus (b) and entorhinal cortex (c) of a control case and in the CA1 region (d), dentate gyrus (e) and entorhinal cortex (f) of an AD patient. Some “ghost tangles” are shown scattered between MPO-immunoreactive amyloid plaques in the CA1 region of an AD case (g). Similar cells on the adjacent section are ir for tau (p-TAU; h). A negative control for the staining protocol is shown in i. Bar 25 μm