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. 2017 Aug 28;8:442. doi: 10.3389/fneur.2017.00442

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Copyright © 2017 Tuem and Atey.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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PregS-stimulated trafficking of functional N-methyl-d-aspartate (NMDA) receptors to the cell surface via a non-canonical G protein-, PLC-, Ca2⁺-, and protein kinase C (PKC)-dependent mechanism. DAG, diacylglycerol; IP3R, IP3 receptor; NMDA, (N)methyl-d-aspartate; PIP2, phosphatidylinositol 4,5-bisphosphate (59).