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. 2017 Jul 29;108(9):1888–1896. doi: 10.1111/cas.13313

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© 2017 The Authors. Cancer Science published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association.

This is an open access article under the terms of the Creative Commons Attribution‐NonCommercial‐NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.

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Staining of sections from six cases with the non‐terminal respiratory unit‐type morphology, including four representative cases of hepatocyte nuclear factor 4‐α (HNF4‐α)‐positive and NKX2‐1/TTF‐1‐mutated adenocarcinomas (two mucinous adenocarcinomas, one acinar adenocarcinoma, and one enteric adenocarcinoma) (left side) and two representative cases of NK2 homeobox 1 (NKX2‐1)/thyroid transcription factor 1 (TTF‐1)‐positive adenocarcinomas (two solid adenocarcinomas) (right side) for H&E, HNF4‐α, NKX2‐1/TTF‐1, Napsin A, caudal‐type homeobox 2 (CDX2), cytokeratin 20 (CK20), and mucin 5AC (MUC5AC).