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. 2017 Aug 18;32(5):865–874. doi: 10.3904/kjim.2016.033

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Copyright © 2017 The Korean Association of Internal Medicine

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/) which permits unrestricted noncommercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 7. — Immunohistochemical staining of the nucleotide-binding oligomerization domain-like receptor (NLR) family members containing the pyrin domain 3 (NLRP3)-inf lammasome in lung tissues after 24-hour stimulation with cigarette smoking extract (CSE) and diesel exhaust particles (DEPs) (×100). (A) NLRP3-inf lammasome expression was increased by CSE and DEPs in both the normal and (B) elastin-induced emphysema groups. NLRP3-inf lammasome expression was suppressed by N-acetylcysteine (NAC) in both the normal and emphysema groups. LPS, lipopolysaccharide.