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. 2017 Apr 6;8(31):50655–50664. doi: 10.18632/oncotarget.16901

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Copyright: © 2017 Liu et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License 3.0 (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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(A and B) Usp5 knockdown suppressed the viabilities in HepG2 cells (A) and Bel7404 cells (B) with 2.0 μM Dox and 10 μM Cis treatment. (C and D) Usp5 overexpression promoted cell growth in the Dox and Cis treated HepG2 cells (C) and Bel7404 cells (D). **P < 0.01.