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. 2017 May 8;52(7):977–983. doi: 10.1038/bmt.2017.79

Table 1. Patient demographics.

Characteristics (N=53) Median (Range)
Age, yrs 17 (3–57)
Body weight, Kg 50 (12–89)
BSA, m2 1.49 (0.56–1.9)
Sex 35 males; 18 females
   
Diagnosis
 Aplastic anemia 40
 Fanconi anemia 13
   
Regimen
 F-araA/Cy 29
 F-araA /Cy/TBI 20
 F-araA /Cy/ATG 4
   
Donor source
 Matched sibling donor 45
 Alternate donor 8
   
Stem cell source
 Bone marrow 2
 Peripheral blood 48
 Not evaluable (died prior to Tx) 3
CD34 cell dose (× 106/kg) 9.8 (1.3–15.0)
   
HLA Match
 <8 6
 ⩾8 47
   
GvHD prophylaxis
 Cyclosporine/Methotrexate 32
 Post Tx Cy 19
 Not Evaluable 2
   
SNP frequency in F-araA metabolic pathway genes
 NT5E 5′-UTR rs2295890G>C WT, GG-33
 Not available=5 Var, GC/CC-15
 CNT3 Exon 6 rs7853758G>A WT, GG-32
 Not available=7 Var, GA/AA-14
 NT5C2 Intron rs4917996A>C WT, AA-17
 Not available=6 Var, AC/CC-30
 hENT1 Exon 1 rs747199G>C WT, GG-37
 Not available=4 Var, GC/CC-12

Abbreviations: ATG=anti-thymoglobilin; Cy=cyclophosphamide; F-araA=fludarabine; SNP=single-nucleotide polymorphism; Var=variant genotype; WT=wild-type genotype; yrs=years.

Demographics of the patients undergoing HSCT with F-araA/Cy based conditioning regimen with or without TBI/ATG.