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. 2017 Jul 22;6(7):e003693. doi: 10.1161/JAHA.116.003693

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© 2017 The Authors. Published on behalf of the American Heart Association, Inc., by Wiley.

This is an open access article under the terms of the Creative Commons Attribution‐NonCommercial‐NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.

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Proinflammatory gene expression in MT1‐MMP (membrane‐type 1 matrix metalloproteinase)–null vascular smooth muscle cells (VSMCs). A, Primary aortic VSMCs isolated from Cre(−)Mmp14 ^F/ ^FA poe ^−/− and SM22α‐Cre(+)/Mmp14 ^F/ ^FA poe ^−/− mice. F‐actin (green), nuclei (blue). B, Representative genes differentially expressed in VSMCs from 2 independent pairs of Cre(−)Mmp14 ^F/ ^FA poe ^−/− and SM22α‐Cre(+)/Mmp14 ^F/ ^FA poe ^−/− mice. C, Gene Ontology (GO) biological processes represented by the genes upregulated in VSMCs isolated from the SM22α‐Cre(+)/Mmp14 ^F/ ^FA poe ^−/− mice.